<?xml version="1.0"?>
<rdf:RDF xmlns:foaf="http://xmlns.com/foaf/0.1/" xmlns:owl="http://www.w3.org/2002/07/owl#" xmlns:rdfs="http://www.w3.org/2000/01/rdf-schema#" xmlns:rdf="http://www.w3.org/1999/02/22-rdf-syntax-ns#" xmlns:dcat="http://www.w3.org/ns/dcat#" xmlns:dct="http://purl.org/dc/terms/" xmlns:adms="http://www.w3.org/ns/adms#" xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:time="http://www.w3.org/2006/time#" xmlns:dcterms="http://purl.org/dc/terms/" xmlns:vcard="http://www.w3.org/2006/vcard/ns#"><dcat:Dataset rdf:about="https://www.hmp.org.tw/?q=dataset/altered-gut-microbiota-and-inflammatory-cytokine-responses-patients-parkinson%E2%80%99s-disease"><dct:title>Altered gut microbiota and inflammatory cytokine responses in patients with Parkinson&#x2019;s disease</dct:title><dct:description><![CDATA[<p>Fecal microbiota communities from 80 patients with PD and 77 age and gender-matched controls were assessed by sequencing the V3-V4 region of the 16S ribosomal RNA gene.</p>
]]></dct:description><dcat:keyword>Fecal microbiota communities</dcat:keyword><dcat:theme>Sample: Multiisolate, Type: 16S Amplicon Sequencing</dcat:theme><dct:identifier>5ab0e0f1-65e8-4cad-bfb3-adbc0dd3f750</dct:identifier><dct:issued>2020-10-22T12:17:37+08:00</dct:issued><dct:modified>2020-10-22T12:22:43+08:00</dct:modified><dct:publisher>(04) &#x570B;&#x7ACB;&#x81FA;&#x7063;&#x5927;&#x5B78;&#x91AB;&#x5B78;&#x9662;&#x5167;&#x79D1; - &#x5433;&#x660E;&#x8CE2;&#x91AB;&#x5E2B;&#x5718;&#x968A;</dct:publisher><dcat:distribution rdf:resource="https://www.hmp.org.tw/?q=dataset/altered-gut-microbiota-and-inflammatory-cytokine-responses-patients-parkinson%E2%80%99s-disease-0"/></dcat:Dataset><dcat:Distribution rdf:about="https://www.hmp.org.tw/?q=dataset/altered-gut-microbiota-and-inflammatory-cytokine-responses-patients-parkinson%E2%80%99s-disease-0"><dct:title>Taxonomic differences of fecal microbiota in PD and control groups</dct:title><dct:description><![CDATA[<p>a Linear discriminant analysis (LDA) effect size (LEfSe) analysis revealed significant bacterial differences in fecal microbiota between the PD (positive score) and control groups (negative score). LDA scores (log10) &gt; 2 and P &lt; 0.05 are shown. b Cladogram using the LEfSe method indicating the phylogenetic distribution of fecal microbiota associated with PD and control participants. c The relative abundance of Prevotella was significantly higher in the control group than in patients with PD. **P &lt; 0.01</p>
]]></dct:description><dct:issued>2020-10-22T12:22:43+08:00</dct:issued><dct:modified>2020-10-22T12:22:43+08:00</dct:modified><dcat:accessURL rdf:resource=""/><dcat:downloadURL rdf:resource="https://www.hmp.org.tw/sites/default/files/12974_2019_1528_Fig3_HTML.jpg"/><dcat:mediaType>image/jpeg</dcat:mediaType><dct:format>jpeg</dct:format><dcat:byteSize>55830</dcat:byteSize><foaf:page>https://www.hmp.org.tw/?q=dataset/altered-gut-microbiota-and-inflammatory-cytokine-responses-patients-parkinson%E2%80%99s-disease-0</foaf:page></dcat:Distribution><foaf:Agent rdf:about="https://www.hmp.org.tw/publisher/n0"><foaf:name>DKAN</foaf:name><foaf:homepage>https://www.hmp.org.tw</foaf:homepage><dct:type rdf:resource="http://purl.org/adms/publishertype/NonProfitOrganisation"/></foaf:Agent></rdf:RDF>
